Arteriosclerosis is a non-inflammatory disease of the arteries, which can thicken and harden the arterial wall, lose its elasticity and narrow the lumen. Arteriosclerosis is a vascular disease that occurs with age, and its regularity usually occurs in adolescence, and becomes aggravated and onset in middle-aged and old age. More men than women, the disease has gradually increased in China and Hong Kong in recent years, becoming one of the leading causes of death in the elderly. The difference between arteriosclerosis and atherosclerosis, strictly speaking, arteriosclerosis is a larger concept. It is a general term for pathological changes in which the arterial wall is thickened, stiffened and inelastic for any reason. Mainly divided into: (1) Small arteriosclerosis.(2) Arterial medial sclerosis.(3) Atherosclerosis – a common type of arteriosclerosis, which mainly leads to myocardial infarction and cerebral infarction. Arterial walls are composed of intima, media, and adventitia. According to the diameter of the tube, the arteries can be divided into three grades: large, medium and small. Large arteries such as the aorta and the media of its large branches contain a large number of elastic fibers, which are very elastic, so they are also called elastic arteries. The middle arteries such as coronary arteries, cerebral arteries, renal arteries and limb arteries contain smooth muscle, so they are also called muscular arteries. Arterioles refer to the arteries with a diameter of less than 1 mm, which belong to the muscular arteries, but the inner elastic membrane is thin and inconspicuous, and there are few smooth muscles in the media.

Blood vessels are the conduits for the transport of human blood. If the blood vessel is blocked, the blood cannot flow smoothly in the blood vessel, which can cause various discomforts in the lightest and life-threatening in serious cases. Early detection of signs of vascular blockage and timely implementation of necessary measures are critical to saving lives. The following symptoms are summarized below. Once they appear, be sure to seek medical attention in time. Sudden chest tightness, alert for pulmonary embolismIf you suddenly feel chest tightness and tightness when you stand up after sitting or lying in bed for a long time, or even faint, you should be alert to the possibility of pulmonary embolism caused by venous thrombosis in the lower extremities and blocking the pulmonary artery. Pulmonary embolism is very dangerous and can lead to sudden death in severe cases. Therefore, if you have the above symptoms, you should go to the emergency department as soon as possible. Chest pain, possibly coronary heart disease, myocardial infarctionCoronary heart disease is called "coronary atherosclerotic heart disease". Coronary arteries are blood vessels that supply blood to the heart. If a blockage or stenosis occurs, the blood supply to the heart will be limited, and severe ischemia will result in chest pain. If it is persistent chest pain, it is even more dangerous, which means that the coronary artery has been completely blocked, the blood flow has been completely interrupted, and the myocardium will be necrotic for more than 15 to 20 minutes, that is, myocardial infarction. Weakness on one side, beware of strokeSudden onset of weakness in one extremity (both upper and lower extremity) suggests a blockage of a cerebral or carotid artery. At the same time, it can also be accompanied by slurred speech, one side of the mouth and eyes, and blurred vision. These symptoms, whether or not they resolve on their own, require immediate medical attention. Eedema and changes in urine output suggest renal artery blockageThe renal artery is responsible for the blood supply of the kidney. If the plaque occurs in the renal artery, it may reduce the blood supply of the renal artery, and even affect the renal function, resulting in symptoms such as fatigue, decreased appetite, changes in urine output, and limb edema. Kidney is an important blood pressure regulating organ, renal artery stenosis can lead to high blood pressure, and the drug control effect of this blood pressure increase is not good. With the aggravation of renal ischemia, its "detoxification" function will be impaired, and even develop into "renal failure", which requires dialysis treatment. Leg pain when walking and blocked lower extremity arteryThe arteries of the extremities are responsible for the blood supply to the upper and lower extremities. In the case of lower extremity vascular ischemia, leg pain may occur when walking, which can be relieved after rest, and the pain reappears when walking again. The medical term is called "intermittent claudication". If the blockage of the lower extremity arteries is ignored and the disease progresses further, it may lead to severe ischemia of the lower extremities, the skin temperature will drop, the pulse of the dorsum of the foot and the posterior tibial artery will weaken or disappear, and the lower extremity pain (ie rest pain) will also develop when not walking. Limbs can rupture, wounds that do not heal, or even necrosis of tissue (gangrene), requiring amputation. Diabetic patients are more likely to develop lower extremity lesions and must pay special attention. Weakened pulse, low blood pressure, clogged upper extremity arteriesThis kind of problem should not be underestimated. If the arteries of the upper extremities are blocked, the pulse of one or both of the arteries will be weakened, and the blood pressure of the arms will decrease. Symptoms of unsteady standing. Asymmetric edema of the lower extremities, beware of deep vein thrombosis of the lower extremitiesIt is easy to occur in patients who are bedridden for a long time, sedentary, dehydrated, tumor, etc., manifesting as swelling and pain in the lower limbs. If it is found that the degree of edema in the two feet or legs is inconsistent, or one side is not swollen and the other side is swollen, it is often a sign of deep vein thrombosis in the lower extremity. The thrombus blocks the veins, causing poor blood flow and causing edema in the limb on which the thrombus formed. At the same time, there will be cold skin, prone to ulcers and other manifestations. Once the thrombus in the veins of the lower extremities breaks off, it will block the pulmonary arteries. To sum up, the main symptoms of vascular blockage include: chest pain, chest tightness, palpitation, shortness of breath, paresthesia, movement or language disturbance, dizziness and headache, blood pressure changes, limb pain, cold hands and feet, swelling of limbs, pain in walking legs, etc. While we are alert to these signs, we must also pay attention to correcting bad living habits, adhere to a healthy lifestyle, and take medicine on time as prescribed by a doctor, or undergo surgery. In June this year, the National Cardiovascular Disease Center released the "China Cardiovascular Disease Report 2016", which is the latest statistics on cardiovascular diseases in the country. The data show that cardiovascular diseases in rural and urban areas account for 45% and 42% of the causes of death respectively, ranking first among all causes of death. Cardiovascular disease accounts for 2 out of every 5 deaths. It is better to nourish blood vessels earlier than to treat them with old age. The "Report" pointed out that the prevalence of cardiovascular disease in my country is on the rise. Among them, there are about 13 million people with stroke, about 11 million people with coronary heart disease, and even 2.7 people with high blood pressure. 100 million. Cardiovascular experts believe that stroke mainly includes 8 risk factors: hypertension, dyslipidemia, diabetes, smoking, atrial fibrillation, overweight, lack of exercise and family history of stroke. The scary thing is that as long as there are any 3 items, it is a high-risk group of stroke. And coronary heart disease also includes high risk factors such as smoking, long-term mental stress, three highs, obesity, lack of exercise, genetic background and long-term use of contraceptives. “Many people think that you only need to worry about blood vessels when you are old. In fact, cardiovascular disease has already 'speeded up'. It is better to take care of blood vessels earlier than to treat them in old age.” He Liqun, Chief Physician of the Department of Cardiology, Wuhan First Hospital It is emphasized that it is best to keep the walls of the blood vessels smooth, soft and elastic before the "scale" in the blood vessels accumulates. This requires us to intervene in terms of lifestyle first. Now that you know the "natural enemies" of blood vessels, such as big fish and big meat, day and night reversal, smoking more, and less exercise, you might as well get rid of them and do more daily maintenance of beneficial blood vessels. Daily nourishing blood vessels to ensure five good habits A famous French doctor once said: "Man and arteries have the same lifespan." By comparing blood vessels to the candles of life, it can be seen that lifespan is closely related to the health of blood vessels. Many experts and professors have repeatedly emphasized the importance of protecting blood vessels in their education to patients. First of all, control sugar and oil in your diet. Now that the pace of life is accelerating, restaurants, takeaways, and even many people's cooking at home are often "concocted" with "high salt, high oil, high sugar" and heavy oil red sauce for the sake of taste, resulting in blood vessel blockage. Therefore, the diet should be light, such as drinking a bowl of multi-grain porridge for dinner, which is conducive to preventing coronary heart disease and controlling cholesterol. Second, exercise to enhance vascular function. Jogging, brisk walking, swimming, various aerobic exercises, gymnastics that help muscles stretch and joints soft, even mild muscle strength exercises and other activities can improve blood circulation, inhibit sympathetic nerve activity, and slow down the heart rhythm in normal conditions. Reduce the burden on the heart. WHO data shows that people over the age of 65 who walk for 4 hours or more per week have a 69% reduction in cardiovascular morbidity and a 73% reduction in mortality compared with those who walk for less than 1 hour per week. Third, ensure sleep. There are not many cases of sudden death due to long-term fatigue, stress, and insufficient rest. Most people recommend that adults fall asleep before 23:00 every day, sleep enough for 7-8 hours, and not be disturbed by anything. Fourth, relax. Sudden mental stress may cause endothelial dysfunction, which impairs the ability of blood vessels to dilate. As a result, blood vessels cannot adjust their function with changes in blood demand, increasing the occurrence of cardiac abrupt events. Fifth, check blood vessels every year and monitor blood pressure. Asymptomatic or uncontrolled high blood pressure is the trigger for many vascular diseases. The risk of cerebral infarction in hypertensive patients is 4-7 times that of normal people. Hypertension not only affects microvascular, but also leads to macrovascular disease. It is necessary to monitor and take medicine on time to keep them within the normal range.

If some signs of arteriosclerosis can be recognized early, and self-protection and monitoring can be strengthened, the speed of its development can be greatly slowed down, and according to the monitoring situation, people can go to the hospital for examination and treatment in time. Those who are worried that they have arteriosclerosis should go to the hospital to check the fundus in time to find the manifestations of retinal arteriosclerosis and achieve the purpose of early diagnosis and treatment. People suspected of having arteriosclerosis should cooperate with PASESA screening - the concept of "preventive treatment". Early detection and early prevention. Confirmed patients with hypertension, hyperlipidemia, diabetes and cardiovascular disease, PASESA cardiovascular measuring instrument should be used with daily medication to understand the condition. Daily cardiovascular testing in critically ill and stroke patients.

The three most important factors for arteriosclerosis are hypertension, hyperlipidemia and smoking. Others such as obesity, diabetes, lack of exercise, nervousness, advanced age, family history, short temper, etc. are quite related. Hypertension: The long-term high pressure of blood impacting the inner wall of the artery causes physical and mechanical damage to the arterial intima, resulting in the easy deposition of blood lipids, the formation of fatty plaques, and the formation of arteriosclerosis and stenosis. Uncontrolled blood pressure will increase the incidence of myocardial infarction by about 2 to 3 times and stroke by about 4 times. Hyperlipidemia: Too much fat in the blood is more likely to deposit on the inner wall of blood vessels to form plaque, resulting in arteriosclerosis and stenosis. Diabetes: People with diabetes will have problems with fat metabolism, and the proteins that transport fat (called lipoproteins) in the blood will be denatured. During the transport process, fat is easily deposited on the inner wall of blood vessels to form fatty plaques. Smoking: The nicotine and carbon monoxide in cigarettes can damage the inner wall of the arteries, and the injured arterial wall will make cholesterol easy to adhere, causing platelets to accumulate and form fatty plaques. At the same time, smoking can also cause coronary artery constriction and spasm, reducing blood flow. Lack of exercise: Exercise can increase high-density lipoprotein and reduce low-density lipoprotein, help the body to excrete excess cholesterol from the biliary tract and intestinal tract, and prevent excess cholesterol from depositing in the lining of blood vessels. In addition, exercise can promote blood circulation, increase the elasticity of blood vessels, lower blood pressure, consume excess calories, reduce the proportion of body fat, increase the proportion of muscle, and lose weight. Therefore, people who lack exercise are prone to atherosclerosis. Obesity: People who are obese or overweight have a higher burden on the heart and a higher chance of abnormal blood fat, thus increasing the risk of atherosclerosis. Obesity can easily lead to hypertension, diabetes, hyperlipidemia and insulin resistance syndrome. Excessive stress: People will increase the secretion of adrenaline due to stress, causing blood pressure to rise, heart rate to increase, and damage to the inner wall of arteries. Family History: Refers to genetic factors. Some people will develop arteriosclerotic disease hereditary in the early stage, and the cause is still unknown. Some people have severe hypercholesterolemia, which accumulates in the blood, thereby promoting arteriosclerosis. Some have early-onset hypertension or are prone to thrombosis. Nutritional causes: A. Risk factors for arteriosclerosis—cholesterol: A large intake of greasy foods and foods rich in cholesterol is the main cause of arteriosclerosis. Cholesterol is an important component of cell membrane, brain and nerve tissue in the body, and is related to the formation of hormones and vitamin D. It has special physiological functions and is indispensable. However, if the intake is excessive, the content of cholesterol in the blood will exceed the normal range, and over time, arteriosclerosis will be induced and endanger health. Clinical studies have found that excessive cholesterol in the blood is one of the important risk factors for coronary arteriosclerosis and cardiovascular disease. B. The fat diet is closely related to arteriosclerosis: the daily diet generally contains fat. If you usually like to eat fatty food without paying attention to the intake of other minerals, too much fat will be deposited on the blood vessel wall and induce arteriosclerosis. and other cardiovascular diseases.

Details the process of atherosclerosis:Atherosclerosis mainly involves large and medium-sized arteries. The etiology and pathogenesis of atherosclerosis are not fully understood, but it has been recognized that high cholesterol, hypertension, and smoking are the main risk factors for the disease. Lipid metabolism disorders, vascular endothelial damage, and platelet adhesion appear as yellow atherosclerosis, so it is called atherosclerosis. The plaque gradually expands, which can narrow and harden the arterial lumen, causing structural and functional changes in tissues and organs. Cholesterol is the most important factor in the development of atherosclerosis. Of the three main risk factors for coronary heart disease (high cholesterol, high blood pressure, and smoking), only high cholesterol is the only necessary prerequisite. Lipoproteins (such as low-density lipoprotein LDL, very low-density lipoprotein VLDL and oxidized low-density lipoprotein Ox-LDL, etc.) that can increase the influx and deposition of cholesterol in the arterial wall can lead to atherosclerosis, Low-density lipoprotein (LDL) usually exists in a non-oxidized state, and the oxidation of LDL will accelerate the occurrence of atherosclerosis. Therefore, preventing [low density lipoprotein] from being oxidized and appropriately reducing [low density lipoprotein] is of great significance for the prevention and treatment of atherosclerosis; Lipoproteins (such as [high-density lipoprotein] HDL, oxidized high-density lipoprotein, etc.) that can promote the export of cholesterol have anti-atherosclerosis. Increasing [high-density lipoprotein] HDL is of great significance. Studies have shown that every 0.5mmol/L increase in HDL can reduce the probability of arteriosclerosis by 20%.Under normal circumstances, the roles of LDL and HDL are in a balanced state. In some hereditary diseases or high-fat diets, the influx and deposition of cholesterol are significantly higher than normal due to a significant increase in the role of atherosclerotic lipoproteins. , resulting in the occurrence of atherosclerosis. When there is more oxidized LDL cholesterol in the body than the body needs, it accumulates on the walls of blood vessels, causing the blood vessels to gradually harden and narrow, but the body does not notice any symptoms for a long time. After a long period of time, the cholesterol accumulated on the blood vessel wall gradually blocked the blood vessels, and the blood flow to the main organs gradually decreased. When organs don't get enough oxygen and nutrients from the blood, they can easily die. A coronary atherosclerotic heart attack occurs when the blood vessels supplying the heart (coronaries) become blocked, causing angina or blockage. If the blood vessels supplying the brain become blocked, a stroke or cerebral hemorrhage can occur.

What is the difference between arteriosclerosis and atherosclerosis? Strictly speaking, arteriosclerosis is a larger concept. It is a general term for pathological changes in which the arterial wall is thickened, stiffened and inelastic for any reason. Mainly divided into three types of large, medium and small arteries, including atherosclerosis, arterial medial calcification and small arteriosclerosis. Among arteriosclerosis, atherosclerosis is the most concerned. It affects the large and medium arteries with considerable consequences. Coronary atherosclerosis can cause insufficient blood supply to the heart muscle, causing angina pectoris and even myocardial infarction. Cerebral arteriosclerosis can cause insufficient blood supply to the brain, leading to dizziness and headache. Bleeding from small aneurysms caused by thrombosis or atherosclerosis in cerebral arteries can lead to stroke, which can cause serious consequences such as loss of consciousness, hemiplegia, and aphasia. Renal arteriosclerosis can lead to high blood pressure. Intestinal arteriosclerosis can cause indigestion, constipation, and diarrhea. Atherosclerosis of the lower extremities can lead to intermittent claudication and, in severe cases, ischemic necrosis of the extremities. The occurrence of arterial medial calcification is age-related. It often occurs in the arteries of the head and neck and limbs of the elderly. There are degenerative pathological changes and deposition in the middle layer of the arteries, resulting in hardening of the blood vessels. But generally does not affect the blood supply, so there is no important clinical significance. Arteriosclerosis occurs in peripheral arterioles. The intima of the arterioles is deposited with amorphous material, which is transparent, resulting in a smaller lumen. This disease is mostly related to high blood pressure. Arteriosclerosis has the greatest impact on the blood supply to the brain and kidneys. From a medical point of view, arteriosclerosis generally refers to lesions caused by atherosclerosis unless otherwise specified.

Central arterial blood pressure (systolic blood pressure, pulse pressure) is closely related to central arterial stiffness. Generally, if central arterial blood pressure is high, central arterial stiffness will also be high. However, the two are different concepts. Different physical meanings should not be confused or substituted for each other. The former is a blood pressure index, and the latter is a mechanical index of the arterial wall. It is possible that both AVI and central arterial blood pressure were high in one patient, while only one measure was high in another. For example, some patients have congenital hypotension with normal central arterial blood pressure, but AVI may be high due to other pathological factors. At present, there is no gold standard for central arterial blood pressure, only estimates.

There are three most dangerous areas of arteriosclerosis in the human body. Cardiac arteriosclerosis, arteriosclerosis of brain tissue, carotid arteriosclerosis. Arteriosclerosis is a systemic disease. However, the degree and impact of arteriosclerosis in different parts of the body are different, and the impact of arteriosclerosis on health in different parts is also different. We know that arteriosclerosis in the heart can lead to myocardial infarction, and arteriosclerosis in the brain can lead to cerebral hemorrhage. What are the consequences of carotid arteriosclerosis? Many people may not yet understand. Arteriosclerosis in the brain. The carotid artery is relatively thick, and the blood directly supplies important organs such as brain tissue and facial features. When the carotid arteries are hardened, it is as if two hands are pinching the neck, resulting in ischemia and hypoxia in the brain tissue. Over time, it can lead to brain atrophy. If the carotid atherosclerotic plaque falls off, it will block the arteries, causing blindness, hemiplegia, and even life-threatening. Therefore, carotid arteriosclerosis will seriously reduce the quality of life of people, and it is indeed one of the most dangerous arteriosclerosis. More recently, cardiologists have studied the causes of carotid arteriosclerosis. The results showed that smoking had the greatest impact on carotid atherosclerosis. Smoking cessation is the best measure to prevent carotid atherosclerosis. If a person does not smoke, or quits smoking in early old age, the risk of carotid atherosclerosis is much smaller.

First understand the difference between central and peripheral blood pressure: The blood pressure measured on the upper arm or wrist with a sphygmomanometer is peripheral arterial blood pressure. Central arterial blood pressure is the blood pressure in the aorta near the exit of the heart. Currently, it cannot be directly measured by non-invasive methods. In clinical practice, invasive measurement of arterial catheter can be used, but it cannot be widely used because of its invasiveness. Due to the phenomenon of arterial system memory in the transmission, reflection and superposition of blood pressure waves, peripheral arterial systolic blood pressure is often higher than central arterial systolic blood pressure. Compression is relatively close. so:1. Compared with peripheral blood pressure, central arterial blood pressure can better reflect the blood pressure of the heart, kidney, brain and other important organs, and has better value for judging organ damage in patients with hypertension.2. In the treatment of hypertension, a large number of clinical studies have shown that beta blockers and vasodilators (such as ACEIs) have little difference in the antihypertensive effect of mean arterial blood pressure, but the former has little effect on the reduction of central arterial systolic blood pressure and pulse pressure. The pressure effect is not good. In particular, there are significant individual differences in the antihypertensive effect of different drugs on central arterial blood pressure. Therefore, there is a consensus in the medical community that it is necessary to monitor the central arterial blood pressure in individual patients in the treatment of hypertension

The heart pumps blood outward. When the blood vessels are softer, the blood pumped by the heart will spread out and return slowly. When the heart is pumping blood during systole, the reflected waves cannot return, so the blood pressure will not be very high. At this time, the load on the heart will not be high, and the blood vessels will store a lot of energy. When the heart is no longer pumping blood, it can push the blood to continue walking. If the blood vessels become very hard, the reflected waves will return very early, and the heart is still pumping. At the time of blood, the reflected wave has already returned, so the heart will have a high load, and the heart will be very tired if you try to push the blood out. Otherwise, the reflected wave will disappear in the diastolic period, and it will drop very quickly. Then the peripheral arteries also need blood supply, especially the coronary arteries. During the diastolic period, the coronary arteries are mainly infused. When blood supply is required, the blood pressure is very low and cannot be supplied, which will cause ischemia. Mainly from the perspective of the load on the heart, So the softer the blood vessels, the better.

Central artery is a rough concept. The pathological and physiological principles of AVI and API are different. The aging of central arteries is faster than that of peripheral blood vessels. The elasticity of central arteries is relatively large. Central arteries and peripheral arteries are mainly structurally different. In the same way, it is mainly composed of elastic mechanism. The elastic mechanism tends to age with age. The stiffness of central arteries is different from that of peripheral arteries. It has become a consensus that central arteries increase with age and hypertension. It is a muscular blood vessel, the central artery is an elastic blood vessel, the heart will be fatigued after 100,000 beats a day, and the central artery will become thicker and thicker (1-2 times) and easy to expand. The two indicators cannot be unified. Stiffness is more valuable in predicting coronary heart disease and stroke than peripheral arterial stiffness. It has been agreed that central arterial stiffness is more important. Currently, evaluating central arterial stiffness is one of the indicators in the medical field of cardiovascular disease. A good method, no one dares to say that he is the gold standard, all are estimates, including Omron's PWV, AVIAPI is one of the effective detection and evaluation methods, we can separate the assessment, one is the stiffness of the central artery, and the other is the upper arm. The local arterial stiffness of the brachial artery cannot be distinguished by other testing instruments. PWV can only measure a comprehensive index of the degree of arteriosclerosis. Our advantage is that we can identify this physiological location. According to the measurement principles of AVI and API, AVI mainly reflects the stiffness of the central artery, while API mainly reflects the stiffness of the brachial artery (belonging to the peripheral artery), thus realizing the separate measurement of the stiffness of the central artery and the peripheral artery, which is consistent with baPWV (actual arterial stiffness). Compared with the combined stiffness of central and peripheral arteries), the physiological significance of the measurement is more clear. AVI can be used as the main evaluation index of arteriosclerosis, and API can be used as auxiliary index. In certain circumstances, such as the presence of specific pathological factors that cause increased peripheral arterial stiffness, APIs can be used for monitoring.

API can only be used as a representative, not all can be tested. API measures the brachial artery, not the lower extremity, but the brachial artery has a certain correlation with the lower extremity. If your peripheral blood vessels are stiff, the stiffness in the next week is very likely. There is also a certain correlation with the brain.

Reduce the intake of fat: You should eat less fried foods with a high content of "saturated fatty acids" and shrimp, liver, kidney and other internal organs and egg yolks containing "high cholesterol" foods. Do not smoke and reduce second-hand smoke: Tobacco damages cardiovascular endothelial cells, damages endothelial system function, can cause cardiac muscle hypertrophy and thickening, affects normal diastolic and systolic movements and reduces HDL. Adhere to a moderate amount of physical activity: Physical activity needs to be determined according to the original physical condition. It should be done gradually. It is not advisable to do strenuous exercise. 10 minutes. Relaxing the mind and body: Chronic depression or constant tension can stimulate sympathetic nerve activation, resulting in increased heart rate, vasoconstriction, increased blood pressure, and reduced blood flow.

Milk: Contains a factor that can reduce the concentration of cholesterol in serum. Milk also contains a lot of calcium, which can also reduce the absorption of cholesterol. Soybeans: Substances that contain saponins that lower blood cholesterol levels. Ginger: Contains an oleoresin, which has obvious effects on lowering blood lipids and cholesterol. Garlic: contains volatile hormones, which can eliminate the fat accumulated in the blood vessels, and has obvious lipid-lowering effect. It has a good effect on the prevention of atherosclerosis and myocardial infarction. Eggplant: It contains more vitamin P, which can increase the elasticity of capillaries and has a certain effect on preventing high blood pressure, arteriosclerosis and cerebral hemorrhage. Agaric: It can lower cholesterol in the blood, lose weight and fight cancer. Oat: It has the effect of lowering blood cholesterol and triglyceride, and eating it regularly can prevent atherosclerosis. Sweet potato: It can supply a large amount of collagen and mucopolysaccharide substances to the human body, which can maintain the elasticity of arterial blood vessels. Hawthorn: It has the effect of strengthening and regulating the heart muscle, increasing the amplitude of cardiac contraction and coronary blood flow, and also reducing serum cholesterol. Tea: Refreshing, strengthening the heart, diuretic, eliminating greasy and lowering lipids. Tea polyphenols in tea can not only remove fat but also significantly inhibit the increase of cholesterol content in plasma and liver, and inhibit the prevention and treatment of arteriosclerosis. Studies have shown that tea pigment has significant anti-coagulation, promoting fibrinolysis, preventing platelet adhesion and inhibiting the proliferation of arterial smooth muscle cells. Sea fish: It has the effect of lowering blood lipids. Studies have shown that people who eat more fish have lower plasma lipids. It has the effect of preventing arteriosclerosis and coronary heart disease. Tangerine: Eating more can improve the detoxification ability of the liver, accelerate the conversion of cholesterol, and reduce the content of serum cholesterol and blood lipids. Panax notoginseng: It has a wide range of pharmacological effects related to dispersing blood stasis and hemostasis, reducing swelling and relieving pain, including hemostasis, antithrombotic, hematopoietic promotion, vasodilator, blood pressure lowering, anti-myocardial ischemia, anti-cerebral ischemia, anti-arrhythmia, Anti-atherosclerosis, anti-inflammatory, hepatoprotective, anti-tumor, analgesic and other effects. Chili peppers: One red pepper contains a daily serving of beta-carotene, a powerful antioxidant that fights the oxidation of low-density cholesterol (LDL) to its harmful form. Once oxidized, LDL, like butter left in the refrigerator, turns into a bad substance that clogs arteries. Antioxidant-rich foods: high-efficiency antioxidant foods such as tomatoes, pomegranates, anthocyanins, astaxanthin, etc., can increase high-density lipoprotein, reduce low-density lipoprotein and remove triglycerides through antioxidants. Prevent arteriosclerosis.

3 times the dosage: Vitamin C Wheat Germ Oil Lecithin 2 times the dosage: deep sea fish oil spirulina flakes 1 times the dosage: protein powder propolis Lecithin: It has the effect of emulsifying and decomposing oil, reducing blood viscosity, reducing the deposition of lipid peroxide in the blood vessel wall, and promoting the dissipation of atherosclerotic plaques. Protein powder: enhance the body's immunity. Propolis: lowers blood sugar concentration, regulates blood pressure in both directions; inhibits a variety of germs and cancer cells, and prolongs life. Spirulina: reduce the amount of cholesterol contained in the human body, regulate blood lipids, and change the acidic constitution. Sacred sea polyene fish oil and deep sea fish oil: The EPA and DHA contained in it can regulate blood lipids, clear blood clots, reduce the concentration of low-density lipoprotein in the blood, increase the content of high-density lipoprotein, and prevent blood clots. Wheat Germ Oil (Beijian Natural Vitamin E): Fight against free radicals in the body, reduce the production of lipid peroxides, and participate in the growth and repair of the intima of arterial blood vessels. Vitamin C: Helps break down triglycerides, convert cholesterol into bile acids, and scavenge free radicals.